1. ^^1 2 3 4 {{cite web|title=4-Methylphenethylamine|url=http://www.chemicalbook.com/ProductChemicalPropertiesCB3299377_EN.htm|work=Chemical Book|accessdate=21 July 2014}}
2. ^¹ {{cite journal |vauthors =Wainscott DB, Little SP, Yin T, Tu Y, Rocco VP, He JX, Nelson DL |title=Pharmacologic characterization of the cloned human trace amine-associated receptor1 (TAAR1) and evidence for species differences with the rat TAAR1 |journal=The Journal of Pharmacology and Experimental Therapeutics |volume=320 |issue=1 |pages=475–85 |date=January 2007 |pmid=17038507 |doi=10.1124/jpet.106.112532 |quote=Several series of substituted phenylethylamines were investigated for activity at the human TAAR1 (Table 2). A surprising finding was the potency of phenylethylamines with substituents at the phenyl C2 position relative to their respective C4-substituted congeners. In each case, except for the hydroxyl substituent, the C2-substituted compound had 8- to 27-fold higher potency than the C4-substituted compound. The C3-substituted compound in each homologous series was typically 2- to 5-fold less potent than the 2-substituted compound, except for the hydroxyl substituent. The most potent of the 2-substituted phenylethylamines was 2-chloro-β-PEA, followed by 2-fluoro-β-PEA, 2-bromo-β-PEA, 2-methoxy-β-PEA, 2-methyl-β-PEA, and then 2-hydroxy-β-PEA.
The effect of β-carbon substitution on the phenylethylamine side chain was also investigated (Table 3). A β-methyl substituent was well tolerated compared with β-PEA. In fact, S-(–)-β-methyl-β-PEA was as potent as β-PEA at human TAAR1. β-Hydroxyl substitution was, however, not tolerated compared with β-PEA. In both cases of β-substitution, enantiomeric selectivity was demonstrated.
In contrast to a methyl substitution on the β-carbon, an α-methyl substitution reduced potency by ∼10-fold for d-amphetamine and 16-fold for l-amphetamine relative to β-PEA (Table 4). N-Methyl substitution was fairly well tolerated; however, N,N-dimethyl substitution was not.}}
3. ^{{cite web | title=4-Methylphenethylamine | url=https://pubchem.ncbi.nlm.nih.gov/summary/summary.cgi?cid=76751 | work=PubChem Compound | publisher = National Center for Biotechnology Information | accessdate=21 July 2014 }}