| 词条 | CCDC104 | |||||||||||||||||||||||||||||||||||||||||||||
| 释义 |
Model organisms
Male and female animals underwent a standardized phenotypic screen to determine the effects of deletion.[4][11] Twenty one tests were carried out on homozygous mutant mice and one significant abnormality was observed: increased susceptibility to Citrobacter infection.[4] References1. ^{{cite web | title = Coiled-coil domain containing 104| url = https://www.ncbi.nlm.nih.gov/gene/112942| accessdate = 2011-12-06}} 2. ^{{cite web |url=http://www.sanger.ac.uk/mouseportal/phenotyping/MBUU/salmonella-challenge/ |title=Salmonella infection data for Ccdc104 |publisher=Wellcome Trust Sanger Institute}} 3. ^{{cite web |url=http://www.sanger.ac.uk/mouseportal/phenotyping/MBUU/citrobacter-challenge/ |title=Citrobacter infection data for Ccdc104 |publisher=Wellcome Trust Sanger Institute}} 4. ^1 2 {{cite journal | doi = 10.1111/j.1755-3768.2010.4142.x | title = The Sanger Mouse Genetics Programme: High throughput characterisation of knockout mice | year = 2010 | author = Gerdin AK | journal = Acta Ophthalmologica | volume = 88 | pages = 925–7 }} 5. ^Mouse Resources Portal, Wellcome Trust Sanger Institute. 6. ^{{cite web |url=http://www.knockoutmouse.org/martsearch/search?query=Ccdc104 |title=International Knockout Mouse Consortium}} 7. ^{{cite web |url=http://www.informatics.jax.org/searchtool/Search.do?query=MGI:4433419 |title=Mouse Genome Informatics}} 8. ^{{Cite journal| last1 = Skarnes |first1 =W. C.| doi = 10.1038/nature10163 | last2 = Rosen | first2 = B.| last3 = West | first3 = A. P.| last4 = Koutsourakis | first4 = M.| last5 = Bushell | first5 = W.| last6 = Iyer | first6 = V.| last7 = Mujica | first7 = A. O.| last8 = Thomas | first8 = M.| last9 = Harrow | first9 = J.| last10 = Cox | first10 = T.| last11 = Jackson | first11 = D.| last12 = Severin | first12 = J.| last13 = Biggs | first13 = P.| last14 = Fu | first14 = J.| last15 = Nefedov | first15 = M.| last16 = De Jong | first16 = P. J.| last17 = Stewart | first17 = A. F.| last18 = Bradley | first18 = A. | title = A conditional knockout resource for the genome-wide study of mouse gene function | journal = Nature | volume = 474 | issue = 7351 | pages = 337–342 | year = 2011 | pmid = 21677750 | pmc =3572410 }} 9. ^{{cite journal | doi = 10.1038/474262a | title = Mouse library set to be knockout | year = 2011 | author = Dolgin E | journal = Nature | volume = 474 | issue = 7351 | pages = 262–3 | pmid = 21677718 }} 10. ^{{cite journal | doi = 10.1016/j.cell.2006.12.018 | title = A Mouse for All Reasons | year = 2007 | journal = Cell | volume = 128 | pages = 9–13 | pmid = 17218247 |vauthors=Collins FS, Rossant J, Wurst W | issue = 1 }} 11. ^{{cite journal|vauthors=van der Weyden L, White JK, Adams DJ, Logan DW | title=The mouse genetics toolkit: revealing function and mechanism. | journal=Genome Biol | year= 2011 | volume= 12 | issue= 6 | pages= 224 | pmid=21722353 | doi=10.1186/gb-2011-12-6-224 | pmc=3218837}} Further reading{{refbegin}}
| last1 = Totland | first1 = C. | last2 = Bredholt | first2 = G. | last3 = Haugen | first3 = M. | last4 = Haukanes | first4 = B. R. I. | last5 = Vedeler | first5 = C. A. | title = Antibody to CCDC104 is associated with a paraneoplastic antibody to CDR2 (anti-Yo) | doi = 10.1007/s00262-009-0742-3 | journal = Cancer Immunology, Immunotherapy | volume = 59 | issue = 2 | pages = 231–237 | year = 2009 | pmid = 19680650 | pmc = }}{{refend}}{{gene-2-stub}} 2 : Human proteins|Genes mutated in mice |
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